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What is the reported incidence of early satiety on a GLP-1 receptor agonist in TRIUMPH-1?

Asked 11 Feb 2025Modified 14 months agoViewed 32k times
24

Stated plainly: early satiety · a GLP-1 receptor agonist · TRIUMPH-1.

I would like help reading this properly rather than being told what conclusion to reach.

I have the full report including the method section, so I can quote specifics if that helps.

What would I need in addition before this supported a decision?

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EH
askedeighty_six_hours16k2711 Feb 2025
Confirming from the other direction: I did the wrong thing and got exactly the predicted outcome. – Dr_Malik_Osei 2 months ago
Is there a reason to prefer the second method over the first, other than cost? – fib4_reader 3 months ago
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5 Answers

Accepted answer first, then by votes
11

Accepted answer

On the detail: the distinction worth making early is between a tolerability problem, which is unpleasant and self-limiting, and a clinical problem, which is neither. They present differently and the thresholds for each are worth writing down before you need them.

Distinguishing an injection-site nodule from an infection: a nodule is firm, non-tender or mildly tender, not warm, not expanding, and appears within a day or two. Cellulitis is warm, tender, expanding, and often accompanied by systemic features. A sterile abscess sits between the two and is fluctuant. Warmth plus expansion plus fever is the combination that stops being a forum question.

Gastrointestinal adverse events, indicative pooled rates

EventActive armPlacebo armTiming
Nausea40–45 %15–20 %Peaks 1–2 wk after each step
Vomiting15–25 %5–8 %Follows nausea
Diarrhoea20–30 %10–15 %Early, variable
Constipation20–25 %8–12 %Later onset, persistent
Discontinuation for GI events4–7 %1–2 %Mostly during escalation

Ranges span agents and doses; read the specific prescribing information for a specific figure.

Fatigue attribution is a subtraction problem. Take out the energy deficit, the dehydration, the electrolyte shortfall and the poor sleep, and what remains attributable to the drug in the trials was modest — placebo-arm fatigue rates were within a few points of active-arm rates in most of the programme. The corollary is that the fixable causes are usually the actual causes.

SURMOUNT-1 reported gastrointestinal events as the most frequent adverse events, mostly mild to moderate and mostly during escalation, with discontinuation for adverse events in the single digits per cent[1].

One qualification — reported incidence in a monitored trial population is a lower bound on what happens in an unmonitored one, because trial participants were escalated to protocol and supported through it.

Most of this resolves. The point of knowing the pattern is to recognise the small fraction that does not.

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LC
answered · acceptedlabel_claim11k182 Mar 2025
8Thank you — the worked example is what makes this usable. – lipid_panel_q 3 months ago
Related: the same reasoning applies to the counter-ion question. – h_pergande 5 months ago
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31

It helps to be literal here: timing is the most useful diagnostic feature here and it is the one most often omitted from the question.

Constipation outlasts the nausea because it has two causes and only one of them resolves. Gastric emptying accommodates over weeks; total intake, and therefore stool volume and the osmotic load reaching the colon, does not recover until intake does. This is why fibre alone can make it worse — you add bulk to a system that is short of water and short of motility.

In practice, the gallbladder signal tracks the rate of weight loss more than it tracks the drug. Rapid mobilisation of adipose tissue increases biliary cholesterol saturation and reduces gallbladder motility; that combination is lithogenic whether the loss came from a drug, a very-low-energy diet or bariatric surgery. The drug contribution on top of that is present but smaller than the rate contribution.

A symptom diary with dates against dose steps answers most of these questions without anyone needing to guess.

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BB
answeredbac_or_bust37k13825 Mar 2025
6

To be exact about it, look at the placebo arm before concluding anything about attribution. The placebo-arm rates for most of these events are not small, because the events themselves are common in the underlying population.

Early satiety is not a side effect; it is the mechanism being observable. The useful distinction is between satiety, where you stop eating without distress, and aversion, where the thought of food is unpleasant. The first is the intended effect. The second frequently precedes the dose being too high or escalated too fast.

Vomiting matters mostly through its consequences. Loss of gastric fluid depletes sodium, chloride and potassium, and hypokalaemia presents as exactly the fatigue and cramping people attribute to the drug. Persistent vomiting also makes a renal panel uninterpretable, because a pre-renal picture looks like renal impairment.

Telogen effluvium following substantial weight loss is documented independently of any pharmacotherapy, which is the cleanest argument that the rate of loss rather than the agent is the primary driver.

Fix the fixable causes first — fluid, electrolytes, sleep, intake — before concluding that the compound is responsible.

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SL
answeredsian_llewellyn85k2488 Jun 2025
3

Mechanically, the honest framing is that this is very common, usually self-limiting, and occasionally the presentation of something that is not self-limiting at all — and the differentiating features are specific enough to be worth knowing.

The telogen effluvium timing signature is the diagnostic feature: hair enters the shedding phase two to four months after the insult, so shedding that starts at month three of rapid loss and peaks around month four to five is the expected pattern. Shedding that starts in week two is not telogen effluvium and warrants a different question. In either case the follicle is not destroyed and regrowth is the rule.

Write down in advance which symptoms mean stop and seek care. It is a short list and it is much easier to write when you are well.

edited 22 Feb 2025 by pk_curve — tightened the wording; no substantive change

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PC
answeredpk_curve12k1519 Feb 2025
3Minor: the trial name is hyphenated in the original publication. – charge_state_3 6 months ago
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3

Stated carefully, the mechanism explains the pattern. Delayed gastric emptying plus central appetite suppression produces early satiety, and early satiety plus a slowed transit produces exactly the symptom cluster people report.

Pancreatitis red flags worth memorising rather than looking up: severe, persistent epigastric pain radiating to the back, worse lying flat and better sitting forward, with nausea and vomiting that does not settle. That combination is an urgent assessment, not a dose adjustment. An isolated lipase elevation without that picture is common and usually not pancreatitis.

If the timing does not fit the escalation, look for another explanation before settling on the drug.

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MH
answeredm_haraldsen38k3814 Mar 2025
5Worth flagging that this changed in 2025, so older answers on the site are out of date. – Dr_Priya_Raghunathan 4 months ago
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Your answer

Ask PeptideStack is a static archive. Posting is closed, but the norms are worth stating: answer the question that was asked, show your working, cite the trial or the certificate, and say plainly where the evidence runs out.

Not medical advice. Research-use-only compounds are not approved for human use.